FCER1G and cytomegalovirus infection: The 3 HCMV seropositive participants in each group were evenly distributed across the CD56dim CD57+NKG2C+ and CD57+FCεR1γ- frequency range suggesting HCMV infection (known to expand populations of highly differentiated and adaptive NK cells13), was not driving the differences observed between the two groups (Supplementary Fig. 4f).