In 2021, Rebelo et al. [2] first identified bi-allelic loss-of-function mutations in the peroxiredoxin 3 (PRDX3) gene, which resulted in the autosomal recessive spinocerebellar ataxia type 32 [SCAR32, Online Mendelian Inheritance in Man (OMIM) #619648], typically presented with non-progressive cerebellar ataxia. The gene discussed is PRDX3; the disease is Progressive cerebellar ataxia.