We aimed at identifying neutrophils (via Ly6G staining), accumulating in psoriatic skin of CD11c-IL-17Aind/ind mice (19), and Th17 cells (via RORγT staining), releasing IL-17A, both key events in the pathomechanism of human plaque psoriasis (13). The gene discussed is ITGAX; the disease is psoriasis vulgaris.