The top five biological functions in GWI include childhood-onset systemic lupus erythematosus (SLE), ATAD3A-related type I interferonpathy, SLE, activation of leukocytes, and antineutrophil cytoplasmic antibody associated vasculitis (all p < 0.0001). This evidence concerns the gene ATAD3A and anti-neutrophil cytoplasmic antibody-associated vasculitis.