IL17A and Epstein-Barr virus infection: Other gammaherpesviruses, including EBV, KSHV, and MHV68, do not encode a viral IL-17A homolog, yet in the context of infectious mononucleosis, a clinical disease associated with recent EBV infection, there is a significant increase in IL-17A-producing CD4+ T cells that persists at least 1 month following the resolution of clinical symptoms (43).