To determine the impact loss of B cell-intrinsic IL-17RA signaling had on the distribution of latently infected cells in the peritoneal cavity at 16 days post-infection, given the decrease in overall viral latency observed there (Fig. 2C) in the absence of B cell-intrinsic IL-17RA signaling, we magnetically sorted CD19-positive B cells from MHV68-infected CD19 Cre-positive and CD19 Cre-negative mice and performed limiting dilution-PCR to determine the frequency of latently infected peritoneal B cells (Fig. 7B) and non-B cells (Fig. 7C). Here, IL17RA is linked to infection.