Co-immunostaining of SLIT2 and MBP revealed increased SLIT2 expression in neurites and its colocalization with MBP-positive axons in tauopathy mice, which was markedly attenuated by Dap12 deletion (Fig. 6E, F, supplementary Fig. 7F-G, supplementary Table 6). This evidence concerns the gene SLIT2 and tauopathy.