KLRC1, KLRD1, PRF1, BIN2, and SLFN5 showed enrichment concentrated in immune-inflammatory modules—including IL-17 signaling, the TNF/NF-κB axis, and innate immune sensor pathways (NOD-, RIG-I-, and Toll-like receptors)—consistent with psoriasis-relevant Th17/innate activation. This evidence concerns the gene TNF and psoriasis.