Specifically, FGF19 promotes the viability and EMT of PCa cells, as demonstrated by increased cell survival, elevated expression of N-cadherin, a marker of EMT, and reduced levels of E-cadherin in PCa cell lines such as LNCaP and PC3 under conditions of androgen deprivation 195 (Figure 5). This evidence concerns the gene FGF19 and posterior cortical atrophy.