TGFB1 and myocardial infarction: Both in vitro and in vivo studies unequivocally demonstrated that ZnSrMo-LDH/Cu-BSA provided excellent cardioprotective effects by modulating various biological mechanisms, including scavenging of ROS, maintenance of calcium homeostasis, inhibition of apoptotic and TGF-β signaling pathways, thereby alleviating myocardial injury caused by MI/R stimulation.