Whilst the role of YAP1 in myofibroblast/myCAF conversion is well-established and whose silencing, similar to TGFβ2 knockdown, impaired cancer cell-induced conversion of Detox-iCAF to ECM-myCAF in vitro [10], this is to our knowledge the first time that interaction of these pathways has been demonstrated in the context of myCAF biology. This evidence concerns the gene TGFB2 and cancer.