In contrast, no significant correlation was found between the phosphorylation of ERK, a downstream target of the Ras/Raf pathway that is often deregulated in melanoma, and neither MCL1 level nor mTORC1 activity (pERK vs MCL1: p = 0.15, pERK vs pS6: p = 0.58). Here, MCL1 is linked to melanoma.