The enrichment results in C2 sets showed that GSK-3β may be related to SARS-CoV-1–modulated host translation machinery, antigen processing and presentation by MHC class II molecules, genes downregulated during cisplatin resistance, allograft rejection, downregulated genes in NK cells between progressive and stable idiopathic pulmonary fibrosis, initial triggering of complement, defensins, fatty acids, and DC pathways. This evidence concerns the gene GSK3B and idiopathic pulmonary fibrosis.