BLOC1S1 and ischemic stroke: (1) Although we have demonstrated that NBP can effectively alleviate mitochondrial dysfunction through inhibiting the ERK‐mediated phosphorylation of Drp1 and impeding GCN5L1 interaction with Drp1, the present study has not been able to demonstrate that the protective effects of NBP against mitochondrial injury rely also on its regulation of GCN5L1‐mediated Drp1 acetylation in ischemic stroke patients.