It is well known that AngII promotes pathological vascular remodeling by inducing VSMC proliferation, migration, hypertrophy, and apoptosis.[24, 25] We next performed an AngII‐induced AIP experiment in mouse VSMCs by coculturing AngII‐treated/untreated VSMCs in a transwell chamber culture system, where AngII‐untreated VSMCs were seeded in the upper chamber and AngII‐stimulated VSMCs in the lower chamber (Figure 1j). This evidence concerns the gene AGT and autoimmune pancreatitis.