HMGB1 and neoplasm: This section focuses on how OV-induced ER stress and reactive oxygen species (ROS) regulate tumor cell immunogenicity, key DAMP (CRT, ATP, HMGB1) mechanisms, and how OV-specific pathways (e.g., PERK/eIF2α-mediated ER stress) complement conventional ICD induction [81,[132], [133], [134]].