Due to the strong proliferation ability of cancer cells, the neovascularization of tumors cannot meet the growth needs of tumor cells, resulting in a large amount of necrosis and a decrease in PPARα expression levels.[39] PPARα agonists can increase the expression level of PPARα in the liver, reduce oxidative stress and inflammatory responses, and alleviate organ damage.[40,41] In our study, QUE group, TET group, and OA group can all upregulate the expression of CAT and PPARα genes, among which the upregulating effects of QUE group and TET group are significant. Here, PPARA is linked to neoplasm.