GAS5 and acute kidney injury: GAS5 (Growth arrest-specific transcript 5) shows context-dependent regulation: in ischemia–reperfusion AKI, GAS5 increases and exerts pro-apoptotic effects by acting as a ceRNA for miR-21 in tubular epithelium, and reviews place GAS5 in lncRNA–miRNA networks integrating apoptosis, oxidative/inflammatory stress, and autophagy in AKI [86,90].