SLC5A2 and myocardial infarction: The study revealed an increase in the expression level of SGLT2 in association with a decrease in the expression of miR-141 during the development of fibrosis after the induction of myocardial infarction in rats via the permanent ligation of the left anterior descending branch of the coronary artery, and SGLT2 knockdown improved the condition of the heart muscle, as did an increase in miR-141 expression [230].