In fact, in cervical cancer cells, PD has been shown to inhibit proliferation: inducing cell cycle arrest at the G0/G1 phase and suppression of metastasis by increasing E-cadherin and decreasing N-cadherin, Snail, and Slug expression, thereby reducing cell migration and invasion and downregulating c-Myc: The proto-oncogene c-Myc, associated with cell proliferation and metastasis, is significantly decreased following PD treatment, contributing to its anticancer effects [14]. The gene discussed is MYC; the disease is cervical carcinoma.