Molecular docking findings indicate that the environmental endocrine-disrupting chemical BPA specifically interacts with key targets essential for the function of renal cell carcinoma cells, including CHRM3, GABBR1, CCR4, KCNN4, PRKCE, CYP2C9, HPGD, and FASN. Here, CCR4 is linked to hereditary clear cell renal cell carcinoma.