In summary, snoRNAs and SNHGs are central regulators of cervical cancer proliferation, apoptosis, and radioresistance through the modulation of p53 stability (e.g., SNORD6) and ceRNA-mediated signaling pathways (e.g., SNHG6/miR-485-3p/STYX axis, SNHG12/miR-148a/CDK1 pathway). This evidence concerns the gene SNHG12 and cervical cancer.