For example, SNHG11, which is upregulated in lung cancer and correlates with poor prognosis, promotes tumor progression by activating the Wnt/β-catenin pathway through two distinct mechanisms: acting as a ceRNA via the SNHG11/miR-4436a/CTNNB1 axis and by directly binding to β-catenin [34]. This evidence concerns the gene SNHG11 and lung cancer.