MOG and autoimmune encephalitis: Immune-mediated cerebellar ataxia (IMCA) associated with neuronal antibodies has only recently been recognized in children, typically occurring as part of the broader neurological manifestations of autoimmune encephalitis (AIE) syndromes, involving antibodies such as N-methyl-D-aspartate receptor (NMDAR), myelin oligodendrocyte glycoprotein (MOG), and glutamic acid decarboxylase 65 (GAD65) [2].