Specifically, fever‐induced HSP70/90 drives macrophage adaptation,[64, 68] T‐cell migration,[74] and Th17 differentiation,[62] whereas IL‐1β/IL‐6/TNF‐α enhances lymphocyte adhesion,[19] Th1‐polarized[72] and leukocyte exhibits promising clinical potential in oncology and infection control. The gene discussed is IL6; the disease is infection.