Studies have found that chemokines such as CCL2, CCL3, and CXCL10 exhibited increased expression during the process of liver damage or liver inflammation [34,35], to which it was mentioned by Brass and Brenndörfer [36] that liver damage could be released by focusing on the recruitment and infiltration of neutrophils, decreased by chemokine receptors. This evidence concerns the gene CCL3 and Hepatitis.