CXCR5 and diabetic neuropathy: Figure 2 shows that CXCL10, CCL2, and CCL7 surge in dorsal-root-ganglion neurons, astrocytes become rich sources of CXCL1, CXCL10, and CXCL12, soluble CX3CL1 activates microglial CX3CR1, and CXCL13 becomes the dominant spinal chemokine, acting on astrocytic CXCR5 in diabetic neuropathy and on neuronal CXCR5 in post-ischaemic pain [23–35].