In addition to swallowing dysfunction, post-stroke immunodeficiency, characterized by elevated levels of interleukin-6 (IL-6), interleukin-10 (IL-10), and C-reactive protein (CRP), along with reduced levels of repulsive guidance molecule A (RGM-A), is increasingly recognized as a key factor contributing to pulmonary vulnerability in post-stroke patients. This evidence concerns the gene CRP and Immunodeficiency.