PLAU and alcoholic liver diseases: KEGG enrichment analysis revealed that the identified proteins were significantly enriched in several pathways (q < 0.05), including fatty acid degradation (hsa00071, q = 0.0049; 3 genes: ADH5, ADH6, ECI2), the NF-κB signaling pathway (hsa04064, q = 0.0341; 3 genes: CXCL12, PLAU, CD14), tyrosine metabolism (hsa00350, q = 0.0420; 2 genes: ADH5, ADH6), alcoholic liver disease (hsa04936, q = 0.0420; 3 genes: ADH5, ADH6, CD14), and pyruvate metabolism (hsa00620, q = 0.0445; 2 genes: ADH5, ADH6) (Figure 5).