Specifically, fasting blood glucose and high-sensitivity troponin (hs-cTnI) were excluded: fasting blood glucose, which reflects metabolic status and may correlate with cardiac energy metabolism in HF, had 28.3% of data missing (115/407 cases) primarily because fasting samples were not routinely collected for critically ill infants; hs-cTnI, a more sensitive marker of myocardial injury than conventional cTnI, had 24.6% missing data (100/407 cases) due to variations in laboratory testing protocols during the 12-year study period. This evidence concerns the gene TNNI3 and hydrops fetalis.