BECs from SE donors exhibited a significantly higher average expression level at d2, with a 1.69-fold increase compared to NSE donors (LM FDR=9.2e-03; Supplemental Table 4) and a 1.92-fold increase compared to HC with significantly different kinetics characterized by sustained higher levels at each post-infection timepoint among SE donors (SE vs NSE GAMM Shape: FDR=3.5e-05; Figure 1D). The gene discussed is ENO2; the disease is infection.