NLRP3 and Myocardial fibrosis: Similarly, in STZ‐induced diabetic model rats and high glucose‐induced H9C2 cells, NLRP3 and IL‐1β expression were significantly increased compared to controls, whereas activation of the NLRP3 pathway modulated cardiomyocyte pyroptosis and improved myocardial fibrosis and cardiac function in DCM [62].