Previously, GDF15 has been consistently reported as being upregulated in response to neurodegeneration (Isik et al. 2024) and associated with elevated risk for several other age‐related diseases (e.g., coronary artery disease, myocardial infarction, cardiometabolic stroke) (Gadd et al. 2024; Isik et al. 2024). This evidence concerns the gene GDF15 and myocardial infarction.