It exacerbates ferroptosis in hepatocellular carcinoma by modulating the miR-362-3 p/MIOX axis [35], contributes to the ferroptosis-enriched microenvironment in glioma [15], and serves as a therapeutic target for ferroptosis induction in non-small-cell lung cancer [46]. The gene discussed is MIOX; the disease is hepatocellular carcinoma.