It can activate the PI3K/AKT signaling pathway, promote the expression of p-AKT, increase the p-AKT/AKT ratio, up-regulate the expression of the anti-apoptotic protein B-cell lymphoma-2, down-regulate the expression of the pro-apoptotic protein Bax protein, inhibit nerve cell apoptosis, reduce cerebral infarction volume and brain water content, thereby treating cerebral infarction (Yang et al., 2020). Here, AKT1 is linked to cerebral infarction.