Similarly, studies by Jiang (2014) [34] and Sun (2020) [35] have highlighted the role of elevated MALAT1 expression in various cancers, including cervical cancer, where it appears to promote proliferation, invasion, and angiogenesis of cervical cancer cells by serving as a precursor for microRNA (miRNA) or as a sponge for miRNA, thus impacting the post‐transcriptional regulation of gene expression [36]. This evidence concerns the gene MALAT1 and cervical cancer.