M2a-type MG are selectively activated and promote immune elimination and tissue regeneration; M2b-type MG are activated by immune complexes mediated through Fcγ receptors and secrete IL-10; M2c-type MG arise from the deactivation of M1-type MG under the influence of glucocorticoids or IL-10, secreting TGF-β and sphingomyelinase (Wang et al., 2018; Hu and Hua, 2018). Here, TGFB1 is linked to myasthenia gravis.