These include many that had been previously identified, such as LUCAT1, DIO3OS, and ANRIL[7], [11], [12], [77], confirming similar patterns of dysregulation in both inflamed and uninflamed IBD tissues; however the majority of our DE lncRNAs had not been previously associated with CD, including 38 of our predicted lncRNAs. This evidence concerns the gene CDKN2B-AS1 and inflammatory bowel disease.