The cecal microbiome of GNX-stressed female mice was significantly rich in genes correlated with “propanoate metabolism,” “mineral absorption,” “cationic antimicrobial peptide (CAMP) resistance,” “Staphylococcus aureus infection,” “ascorbate and aldarate metabolism,” “porphyrin metabolism,” “atrazine degradation,” “pinene, camphor, and geraniol degradation,” and “tyrosine metabolism,” while 16 significantly abundant functions were enriched in the cecal microbiome of control counterpart including “GABAergic synapse” (Fig. S6). The gene discussed is CAMP; the disease is staphylococcus aureus infection.