(Figure 6G), highlighting that DO promoted the healing of incised wounds by preventing the inflammatory responses and playing its anti‐infection function on one hand, and by facilitating the proliferation and migration of fibroblasts and keratinocytes to enable tissue remodeling by regulating cytokine–cytokine receptor interaction, TNF signaling pathway, NOD‐like receptor signaling pathway and TGF‐beta signaling pathway, et al. This evidence concerns the gene TGFB1 and infection.