This level of misregulation was comparable to that observed for BIN1 exon 11 (13.1 ± 3.7%) and DMD exon 78 (14.5 ± 3.5%), two well‐characterized MBNL regulated alternative splices and known to be deregulated in DM1 (Figure 1d) [6, 7]. The gene discussed is DMD; the disease is myotonic dystrophy type 1.