A well-characterized axis is the lncRNA nuclear enriched abundant transcript 1 (NEAT1)-miR-9-5p-transferrin receptor (TFRC)/glutamic-oxaloacetic transaminase 1 (GOT1) axis in sepsis-associated encephalopathy (SA-E), where sepsis triggers elevated expression of exosome-derived NEAT1 in serum, which competitively binds miR-9-5p, thereby relieving its inhibitory effect on TFRC and GOT1 [77]. This evidence concerns the gene NEAT1 and Sepsis.