For example, abnormal activation and apoptotic imbalance of immune cells in sepsis may be related to its regulation of the mitotic checkpoint, while dysregulation of the SHH/WNT pathway may further exacerbate endothelial barrier damage or organ repair disorders, suggesting the potential role of MYO10 in sepsis-induced multiple organ dysfunction (46–48). This evidence concerns the gene MYO10 and Sepsis.