Studies have reported the emergence of influenza A(H9N2) viruses with an enhanced receptor binding preference for human‐like receptors [33] with the capacity to bind to both α,2‐3 linked sialic acid (avian‐like) and α,2‐6 sialic acid linked receptors (most prevalent in the human upper respiratory tract) [34] although no neutralizing antibodies to influenza A(H9N2) virus were detected indicating asymptomatic or mild infections with influenza A(H9N2) virus [35]. The gene discussed is IGKV6-21; the disease is infection.