TGFB3 and Myocardial fibrosis: The analysis revealed enriched pathways includedECM-receptor interaction and TGF-β signaling pathways, knownfor its role in myocardial fibrosis. Keyproteins such as fibronectin 1 (FN1), laminin subunits (LAMA1 andLAMC2), and transforming growth factor beta 3 (TGFB3) indicated fibrosisand activation of TGF-β/SMAD signaling pathway.−,  Consistent with ECM-related alterations, the focal adhesion pathwaywas also enriched.