Additionally, HBQ1, a component of hemoglobin, may be involved in oxidative stress or inflammation regulation,[44,45] while C10orf32 has mainly been studied in the context of malnutrition and motor dysfunction caused by its deficiency.[46,47] Recent studies have linked high expression of C10orf32 with muscle wasting,[48] but its role in bone metabolism warrants further investigation. This evidence concerns the gene HBQ1 and nutritional deficiency disease.