In addition, high levels of PCs, a major component of the phospholipid bilayer membrane, are required for tumor cell growth and proliferation.[30] We then found that knockdown of TRIM15 in EAC cells reduced the abundance of PCs in the cells, whereas simultaneous knockdown of TRIM15 and YY2/FOXRED1 in the cells reversed the PCs‐reducing effect of knockdown of TRIM15 alone (Figure S11A–F, Supporting Information). The gene discussed is FOXRED1; the disease is neoplasm.