Employing GSEA using gene sets for cerebellar disorders, as previously examined in human fetal cerebellum (Aldinger et al., 2021), and a list of CAS-risk genes (Eising et al., 2019; Hildebrand et al., 2020; Kaspi et al., 2022), we found significant enrichment for genes associated with specific disorders including SCA, ASD and intellectual disability in the FOXP2+ cerebellar organoid population (Fig. 5E; Table S8). Here, FOXP2 is linked to cerebellar disorder.