In MM pathogenesis, primary chromosomal translocations predominantly involve partners such as CCND1, FGFR3/MMSET, MAF, CCND3, and MAFB, while secondary translocations frequently affect MYC, MAP3K14, NFKB1, IL16, IL2RA, IKBKE, TXNDC5, NSD2, and members of the cyclin D family (CCND1‐3) [8, 9]. This evidence concerns the gene MYC and Miyoshi myopathy.