Additional immune cells are also implicated, including neutrophils, early infiltrators that generate ROS and neutrophil extracellular traps [69], NK cells, which produce IFNγ and contribute to adipose inflammation [70], innate lymphoid cells, which secrete IL-17A and IL-22 [71], and B cells, which are increased in obesity and support local antibody production and cytokine secretion [72]. This evidence concerns the gene IL17A and obesity due to melanocortin 4 receptor deficiency.