Mayassi et al. [21] showed the loss of interaction between BTNL3/BTNL8 heterodimers and the duodenal Vγ4+ γδ T cells as a characteristic of active CeD in a study of 62 active CeD, 57 gluten-free diet (GFD)-treated CeD, and 99 control participants. The gene discussed is BTNL8; the disease is cranioectodermal dysplasia.