PPRC1 and myeloid sarcoma: (iv) The ability of PrC-210 to efficiently scavenge and quench ROS, and its association here with 62% suppression of MS severity, clearly supports the basic mechanistic role that ROS toxicity has been assumed to play in human MS etiology, and (v) the 62% PrC-210 suppression of EAE-MS paralysis severity seen here in a “treatment” regimen implies that similar efficacy could be achieved in human MS patients.